Suffering because of Diabetic KetoAcidosis Case Study

Suffering because of Diabetic KetoAcidosis - Case Study ExampleThi study stresses that insulin deficiency as seen in figure 1 DM compelsthe body to breakdownamino acidsandtriglyceridesas opposed to glucose for energy production. Due to high breakdown of glyceride and amino acids, the levels of serum foreswear fatty acids and glycerol goes abnormally leading to high level of uncontrolled lipolysis. The Free Fatty Acids (FFA) rises substantially in the blood meanwhile, muscles lysis goes up. Due to production of acetoacetic acids and hydroxybutyric acids, both of which are strong organic acids increases leading to metabolic acidosis. Metabolic acidosis is one of the classical symptoms of DKA during the initial stage. Because of increase metabolic acidosis, the patients physiologic compensation takes over. In roll to restore normal pH, there is Kausmal respiration that attempt to increase the expiration. Many patients will present with acetone-breathe. Hyperglycemia arises due to in sulin insufficiency leading to high gelt levels in the blood and osmotic diuresis that leads to firing of urinary function. This is a striking similarity with the case study presented. In addition, the urinary excretion of ketones causes increased loss of sodium, potassium and water is lost at large amounts leading to reduced urinary excretion as seen in the patient. As a result of increased loss of electrolytes, potassium often migrates into extracellular component leading to increased potassium levels, often driven back to intracellular by insulin therapy. In install to diagnose DKA, first the patient history and physical assessment helps in identifying which laboratories test to be done. Clinical diagnosis depends on three key lab findings the arterial blood pH normally less than 7.30 with an anion gap of more than 12. Secondly, serum ketones levels are an important diagnostic measurement. The presence of serum ketones and urine ketones portend definitive diagnosis.